By Roger S. Kirby
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Additional resources for An Atlas of Erectile Dysfunction, Second Edition (Encyclopedia of Visual Medicine)
Three major mechanisms are involved: (1) Influx of extracellular calcium through voltageregulated channels; (2) Activation of membrane-bound receptors which allow extracellular calcium to enter through receptor-operated channels; (3) Activation of signal pathways which allow intracellular release of calcium from the sarcoplasmic reticulum. Relaxation of cavernosal smooth muscle may be thought of as ‘resetting’ the contractile machinery. This is mainly accomplished by lowering intracellular calcium.
6′92 ISBN 0-203-00892-8 Master e-book ISBN ISBN 1-84214-241-0 (Print Edition) First published in 2004 This edition published in the Taylor & Francis e-Library, 2005. uk. No part of this book may be reproduced in any form without permission from the publishers except for the quotation of brief passages for the purposes of review Composition by The Parthenon Publishing Group Page 5 Contents Foreword 7 Preface 9 Section 1 A Review of Erectile Dysfunction 11 Section 2 Erectile Dysfunction lllustrated 47 Index 107 Page 6 This page intentionally left blank.
Compounds such as finasteride, which inhibit the activity of 5α-reductase type II, result in shrinkage of the prostate by 20–30%, but have been reported to cause erectile dysfunction in only around 3–5% of patients. However, in the 4-year placebo-controlled study of finasteride recently reported by McConnell and colleagues, nearly 14% of patients taking the active drug experienced some form of sexual dysfunction. Prolactin, which is released from the pituitary gland, acts as an inhibitory factor in male sexual function.